Detailseite
Projekt Druckansicht

Lungenalterung: Einfluss der zellulären Seneszenz auf Lungenentzündung und Reparatur bei Endotoxin- und Klebsiella pneumoniae-induziertem Lungenschaden

Fachliche Zuordnung Klinische Infektiologie und Tropenmedizin
Anatomie und Physiologie
Förderung Förderung von 2019 bis 2024
Projektkennung Deutsche Forschungsgemeinschaft (DFG) - Projektnummer 427406675
 
Erstellungsjahr 2023

Zusammenfassung der Projektergebnisse

The most relevant new findings of our study are: Old mice show a dysregulated activation of the Nlrp3/caspase-1/IL-1β axis in acute lung injury (ALI) that supports the development of neutrophilic pleuritis in old mice compared to young mice upon K. pneumoniae infection. The deregulatory effects were attributable to age-related alterations in the bone marrow rather than the lung, supporting a role for hematopoietic inflamm-aging in lung infections of the elderly. Short chain fatty acids, important metabolites of the gut microbiota, have a beneficial role in the gut-lung axis of the aging organism by reducing pulmonary inflamm-aging and ameliorating enhanced severity of ALI in old mice. The results furthermore emphasized the importance of the gut microbiome and its metabolites in healthy aging beyond the gut. ALI promotes surfactant dysfunction and cellular senescence in alveolar epithelial type II (ATII) cells in old mice. Testing the impact of ATII cell senescence on lung injury and regeneration revealed, however, that ATII cell senescence contributes to and affects regeneration after injury, but is not the main driver of enhanced severity of ALI in old mice.

Projektbezogene Publikationen (Auswahl)

 
 

Zusatzinformationen

Textvergrößerung und Kontrastanpassung