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Projekt Druckansicht

Thrombozyten-Monozyten-Interaktionen bei Systemischen Lupus Erythematodes

Fachliche Zuordnung Hämatologie, Onkologie
Förderung Förderung von 2019 bis 2022
Projektkennung Deutsche Forschungsgemeinschaft (DFG) - Projektnummer 429657170
 
Erstellungsjahr 2023

Zusammenfassung der Projektergebnisse

I have shown that platelet:monocyte interaction shapes the monocyte transcriptome and phenotype in thromboinflammation. Consistent with a proinflammatory platelet effector role, MPA were increased in patients with COVID-19. RNA-Seq revealed a thromboinflammatory monocyte transcriptome upon incubation with platelets. Monocytes aggregated to platelets expressed higher CD40 and tissue factor than monocytes without platelets (p<0.05 for each). Inhibition with P-selectin (85% reduction) and PSGL1 (87% reduction) led to a robust decrease in MPA. P2Y12 and PAR1 inhibition lowered MPA formation (30% and 21% reduction, p<0.05, respectively) and decreased monocyte CD40 and TF expression while GP IIb/IIIa and COX1 inhibition had no effect. Pretreatment of platelets with P2Y12 inhibitors reduced the expression of platelet-mediated monocyte transcription of proinflammatory SOCS3 and OSM. While the COVID-19 pandemic temporarily interrupted my main research project, the finding that infected patients developed a substantial risk of thromboembolic adverse events fueled additional scientific projects in which I critically contributed to analyzing the effector role of platelets on endothelial cells. Here, we could demonstrate that COVID-19 platelets are proinflammatory, exhibit upregulation of inflammatory pathways, and induce endothelial dysfunction.

Projektbezogene Publikationen (Auswahl)

 
 

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