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Regulation of kinetochore function in yeast by modification of the core region of the centromeric nucleosome

Subject Area General Genetics and Functional Genome Biology
Term from 2019 to 2024
Project identifier Deutsche Forschungsgemeinschaft (DFG) - Project number 424365872
 
Final Report Year 2025

Final Report Abstract

Centromeres are the chromosomal regions where kinetochores assemble, forming a physical bridge between DNA and microtubules. During mitosis and meiosis, microtubules pull chromosomes apart, ensuring their accurate segregation to the daughter cells. Errors in this process can result in aneuploidy, contributing to diseases such as cancer and infertility. In this project, we used the yeast centromere as a model to uncover novel mechanisms of centromere regulation. We identified and characterized protein modifications on centromeric proteins that influence the stability of the centromeric nucleosome. We found that these defects in nucleosome stability can be alleviated by strengthening interactions among outer kinetochore proteins, revealing an unexpected functional link between centromeric chromatin and microtubule-proximal kinetochore components. Additionally, we discovered an unexpected mode of centromere regulation involving sugar moieties that may be covalently attached to centromeric proteins. This finding suggests a potential mechanism by which the cell's nutritional state may influence cell cycle progression and chromosome segregation. Finally, we identified a new role for the histone chaperone Yta7 in regulating levels of the centromerespecific histone variant. Our results suggest that Yta7 functions as a hexameric ring that unfolds the histone variant and delivers it to a dedicated deposition machinery for incorporation into centromeric chromatin. This mechanism provides insight into the function of the human homolog ATAD2, which is frequently overexpressed in cancer.

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