Project Details
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The tyrosin kinase Syk: A potential therapeutic target in atherosclerosis

Subject Area Cardiology, Angiology
Term from 2017 to 2024
Project identifier Deutsche Forschungsgemeinschaft (DFG) - Project number 366904753
 
Final Report Year 2024

Final Report Abstract

Like many other diseases, atherosclerosis stems from chronic inflammation of the vessel wall. This is initiated and entertained by various traditional and non-traditional risk factors, varying in response according to genetic background. Our group early on identified SYK as a potential therapeutic target. Here, we demonstrate that SYK indeed limits de novo atherogenesis and favorably alters early plaque phenotype. However, SYK failed to modulate preestablished, more advanced lesions in our hands. Also, SYK was not associated with disease or risk in the LURIC collective interrogated by high dimensional scRNAseq. However, with this project, we identify several cellular biomarkers and associated pathways that correlate both with coronary heart disease and long-term mortality. Our findings may have a profound impact on future risk stratification. Also, we demonstrate significant differences in the immune portfolio of murine and human atherosclerosis, potentially explaining why so many therapeutic endeavors succeed in mice but subsequently fail in humans.

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