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Understanding the role of divergent lncRNA-TF pairs in regulating embryonic cell-fate decisions

Subject Area Developmental Biology
Term from 2018 to 2023
Project identifier Deutsche Forschungsgemeinschaft (DFG) - Project number 400875225
 
Final Report Year 2024

Final Report Abstract

In humans, protein-coding genes are often accompanied by bidirectionally transcribed, divergent non-coding RNAs. However, the relevance of this phenomenon remains poorly understood. Using an hESC-based cardiac differentiation model, we define a class of divergent lncRNAs, yin yang lncRNAs (yylncRNA), that mirror the cell-type specific expression pattern of their protein-coding counterparts. yylncRNAs are preferentially encoded from the genomic loci of key developmental cell-fate regulators. Most yylncRNAs are spliced, polyadenylated transcripts showing comparable expression patterns in vivo in mouse and in human embryos. Signifying their developmental function, the key mesoderm-specifier BRACHYURY (T) is accompanied by YYLNCT, which localizes to the active T locus during mesoderm commitment. Mechanistically, YYLNCT binds the de novo DNA methyltransferase DNMT3B, inhibiting its activity at the T locus during mesoderm differentiation. Consistent with a transcriptspecific function, depletion of YYLNCT disrupts the activation of the T locus, thereby abolishing mesoderm commitment. Collectively, we report a lncRNA-mediated regulatory layer safeguarding embryonic cell-fate transitions.

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